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Original articles
David, C. et al. Gain-of-function human UNC93B1 variants cause systemic lupus erythematosus and chilblain lupus. J. Exp. Med. https://doi.org/10.1084/jem.20232066 (2024)
Al-Azab, M. et al. Genetic variants in UNC93B1 predispose to childhood-onset systemic lupus erythematosus. Nat. Immunol. https://doi.org/10.1038/s41590-024-01846-5 (2024)
Related article
Rael, V. et al. Large-scale mutational analysis identifies UNC93B1 variants that drive TLR-mediated autoimmunity in mice and humans. J. Exp. Med. https://doi.org/10.1084/jem.20232005 (2024)
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Webster, H. UNC93B1 variants promote SLE via TLR activation. Nat Rev Rheumatol (2024). https://doi.org/10.1038/s41584-024-01147-z
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DOI: https://doi.org/10.1038/s41584-024-01147-z