Figures, tables and video
From the following article
Robert Lee and Ravinder Mittal
GI Motility online (2006)
doi:10.1038/gimo75
Figure 1
Distribution of acid reflux times in patients with nonerosive esophageal reflux disease (NERD), erosive esophagitis (EE), and Barrett's esophagus (BE).
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Figure 2
Proposed algorithm for defining gastroesophageal reflux disease based on endoscopic findings and the results of pH studies.
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Figure 3
Schematic of visceral pain. Visceral pain is mediated by visceral afferents that are processed in the dorsal root ganglion.
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Figure 4
Latency of cortical responses to painful esophageal stimuli based on neuroanatomy and gender.
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Figure 5
Esophageal wall thickness in patients with chest pain and controls.
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Figure 6
Esophageal pH (a), manometric pressure (b), and esophageal wall thickness (c) as measured by high-frequency intraluminal ultrasound.
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Figure 7
Simultaneous readings of esophageal pH, muscle layer thickness, and manometric pressure in patients with heartburn.
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Figure 9
VR1 activation in nerve endings triggers the release of substance P (SP), calcitonin gene-related-peptide (CGRP), and neurokinin A.
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Figure 10
Acid-sensing ion channels (ASIC) function as proton-gated channels.
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Figure 11
Anion-sensing ion channel 1 (ASIC1) is inhibited by amiloride and its derivatives.
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Figure 12
The effect of ketamine on acid-induced pain thresholds in the proximal esophagus and the foot.
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