Abstract
Boy land, Wallace and Williams1 have shown that the activity of urinary β-glucuronidase is increased in patients suffering from bladder cancer and suggested that ‘spontaneous’ urinary bladder cancer could be caused by unmasking carcinogens which were inactive as β-glucuronides, but active when liberated by β-glucuronidase in the urine.
This is a preview of subscription content, access via your institution
Access options
Subscribe to this journal
Receive 51 print issues and online access
$199.00 per year
only $3.90 per issue
Buy this article
- Purchase on Springer Link
- Instant access to full article PDF
Prices may be subject to local taxes which are calculated during checkout
Similar content being viewed by others
References
Boyland, E., Wallace, D. M., and Williams, D. C., Brit. J. Cancer, 9, 62 (1955).
Prates, M. D., and Gillman, J. S., S. Afr. J. Med. Sci., 24, 13 (1959).
Boyland, E., Gasson, J. E., and Williams, D. C., Brit. J. Cancer, 11, 120 (1957).
Lewis, F. J. W., and Plaice, Constance H. J., Nature, 184, 1249 (1959).
Author information
Authors and Affiliations
Rights and permissions
About this article
Cite this article
FRIPP, P. Schistosomiasis and Urinary β-Glucuronidase Activity. Nature 188, 507–508 (1960). https://doi.org/10.1038/188507a0
Issue Date:
DOI: https://doi.org/10.1038/188507a0
This article is cited by
-
Über die Enzymurie bei nephrotischem Syndrom
Klinische Wochenschrift (1965)
Comments
By submitting a comment you agree to abide by our Terms and Community Guidelines. If you find something abusive or that does not comply with our terms or guidelines please flag it as inappropriate.